Chemokine receptor CCR2 is not essential for the development of experimental cerebral malaria.

نویسندگان

  • Elodie Belnoue
  • Fabio T M Costa
  • Ana M Vigário
  • Tatiana Voza
  • Françoise Gonnet
  • Irène Landau
  • Nico Van Rooijen
  • Matthias Mack
  • William A Kuziel
  • Laurent Rénia
چکیده

Infection with Plasmodium berghei ANKA induces cerebral malaria in susceptible mice. Brain-sequestered CD8(+) T cells are responsible for this pathology. We have evaluated the role of CCR2, a chemokine receptor expressed on CD8(+) T cells. Infected CCR2-deficient mice were as susceptible to cerebral malaria as wild-type mice were, and CD8(+) T-cell migration to the brain was not abolished.

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عنوان ژورنال:
  • Infection and immunity

دوره 71 6  شماره 

صفحات  -

تاریخ انتشار 2003